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Amiodarone may be continued in patients with amiodarone-induced thyroiditis treated with prednisone
Clinical Thyroidology    Share    Share on FacebookTwitterShare on LinkedinE-mail article
Amiodarone causes thyrotoxicosis in two ways. First, it provides a large iodide load that can induce hyperthyroidism in susceptible individuals with underlying Graves’ disease or multinodular goiter - called type 1 amiodarone-induced thyrotoxicosis. Second, it can cause thyrotoxicosis through a destructive effect on thyrocytes, a form of thyroiditis called AIT type 2. The treatments for AIT usually require discontinuation of amiodarone, in addition to other measures. The aims of the present study were to demonstrate the feasibility of continuing to administer amiodarone in AIT type 2, and to determine whether perchlorate was useful for its treatment. More

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The United Arab Emirates lead the way in the fight against IDD in the Gulf States
ICCIDD    Share    Share on FacebookTwitterShare on LinkedinE-mail article
Experts from ICCIDD emphasized that the UAE has achieved excellent results over the previous decade and is now qualified to apply for a declaration that the country is free of IDD. This was announced during a seminar organized by the health policies sector in the Ministry under the patronage of Abdul Rahman Al Owais, Minister of Culture, Youth and Community Development, and acting Minister of Health. More

Subclinical hyperthyroidism and subclinical hypothyroidism increase the incidence of heart failure in older persons
Clinical Thyroidology    Share    Share on FacebookTwitterShare on LinkedinE-mail article
In many studies, subclinical thyroid dysfunction has been associated with cardiovascular disease, but there is still controversy about these associations. The purpose of this study was to examine the association between subclinical thyroid dysfunction and adjudicated cardiovascular events in a large prospective study of older people with cardiovascular risk factors or preexisting CVD. More

Only a few children with subclinical hypothyroidism progress to overt disease within 3 years
Clinical Thyroidology for Patients    Share    Share on FacebookTwitterShare on LinkedinE-mail article
Overt hypothyroidism is characterized by lab tests that show an elevated TSH and low thyroid hormone levels. The most common cause of hypothyroidism in the U.S. is Hashimoto's thyroiditis, where patients develop thyroid antibodies that attack and destroy the thyroid. Hypothyroidism is treated by replacing thyroid hormone with Levothyroxine. Subclinical hypothyroidism is a mild form of hypothyroidism where the only abnormal hormone level is an increased TSH – the other thyroid hormone levels are normal. More

The Venezuelan iodized salt program provides just the right amount of iodine for both children and pregnant women in Trujillo state
ICCIDD    Share    Share on FacebookTwitterShare on LinkedinE-mail article
Historically, IDD was a severe public health problem in the mountains of Venezuela, as it was throughout the Andean Region. Venezuela has a centralized IDD Control Program that focuses on salt iodization, and includes activities of investigation and monitoring, control and vigilance, as well as communication and education. More

A new form of congenital hypothyroidism with normal serum TSH values has been reported
Clinical Thyroidology    Share    Share on FacebookTwitterShare on LinkedinE-mail article
Thyroid hormone receptors are encoded by two genes that undergo alternative splicing. The known subtypes of thyroid hormone receptors are TRα1 (predominantly present in bone, the gastrointestinal [GI] tract, myocardium, and the central nervous system [CNS]), TRβ1 is predominant in hepatic and renal tissue and TRβ2 in the hypothalamus, pituitary, cochlea, and retina. Thyroid hormone resistance is a rare congenital disease (present in 1 in 40,000 total population), due, in most cases, to dominant negative mutations of the TRβ gene. In a few cases, the gene defect could not be identified. More


 
ATA Thyroid Weekly News Briefs
ATA does not develop, exert any editorial or other control, or guarantee the accuracy, completeness, efficacy, or timeliness, of the materials, information, advertising or promotional activities ("Content") in this publication. Inclusion of Content in this publication does not constitute or imply endorsement, agreement, recommendation, or favoring by ATA of such information or the entities mentioned or promoted herein. Use of any Content appearing or referenced in this publication or obtained from advertisers is voluntary, and reliance on it should only be undertaken after an independent review by qualified experts. ATA is not responsible for, and expressly disclaims all liability for, damages of any kind arising out of use of, reference to, reliance on, or performance pursuant to such Content.

Colby Horton, Vice President of Publishing, 469.420.2601
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Julie Bernhard, Sr. Content Editor, 469.420.2647   
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